ICGA
Active member
If an s1 generation has 4 phenotypes how many phenotypes are in an s2 generation?
Also for example would this be different for an s1 of a polyhybred or supperpolyhybred VS. an s1 of an F1 line etc.
Also does anyone know if all 4 phenotypes from the s1 generation are identical genotypes; what I mean is if you take 100 s1 seeds and separate them into the 4 groups of the 4 phenotypes, will all the seedlings in each group be genetically identical, or just generally phenotypically identical?
Really what I want to know is this; during mitosis when the chromosomes split up does the deck shuffle on an epigenetic level because of "helixing" or does half of "chromosome a" from parent A always unzip and match with the half of "chromisome a" from parents B; Obviously this is the case to some extent as the sequences for a particular trait are supposed to line up some being dominant, recessive etc. But what about the so called "junk DNA" containing epigenetic information and latent genetic contingency. Or genetic contingency that is still forming out of the superstructure of "junk DNA" ?
Another even further out question is this; how does recent data suggesting nonrandom mutations are emerging based on the specific kind of adversity in the environment inform us about genetic drift? Does this actually mean genetic drift can be directed by creating a specific kind of environmental adversity, which would trigger the plant to "have to" mutate a specific way in order to adapt to the intentionally created adversity?
Also for example would this be different for an s1 of a polyhybred or supperpolyhybred VS. an s1 of an F1 line etc.
Also does anyone know if all 4 phenotypes from the s1 generation are identical genotypes; what I mean is if you take 100 s1 seeds and separate them into the 4 groups of the 4 phenotypes, will all the seedlings in each group be genetically identical, or just generally phenotypically identical?
Really what I want to know is this; during mitosis when the chromosomes split up does the deck shuffle on an epigenetic level because of "helixing" or does half of "chromosome a" from parent A always unzip and match with the half of "chromisome a" from parents B; Obviously this is the case to some extent as the sequences for a particular trait are supposed to line up some being dominant, recessive etc. But what about the so called "junk DNA" containing epigenetic information and latent genetic contingency. Or genetic contingency that is still forming out of the superstructure of "junk DNA" ?
Another even further out question is this; how does recent data suggesting nonrandom mutations are emerging based on the specific kind of adversity in the environment inform us about genetic drift? Does this actually mean genetic drift can be directed by creating a specific kind of environmental adversity, which would trigger the plant to "have to" mutate a specific way in order to adapt to the intentionally created adversity?
